FB2024_03 , released June 25, 2024
Allele: Dmel\ocotd-YH13
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General Information
Symbol
Dmel\ocotd-YH13
Species
D. melanogaster
Name
FlyBase ID
FBal0032308
Feature type
allele
Associated gene
Dmel\oc
Associated Insertion(s)
Carried in Construct
Also Known As
otdYH13, otdYH, ocYH
Key Links
Allele class
Mutagen
Nature of the Allele
Allele class
Mutagen
ethyl methanesulfonate
(Royet and Finkelstein, 1995, Wieschaus et al., 1992, Finkelstein and Perrimon, 1990, Finkelstein et al., 1990, Tearle and Nusslein-Volhard, 1987)
Progenitor genotype
Cytology
Description
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
 
UniProtKB/Swiss-Prot
    UniProtKB/TrEMBL
      Expression Data
      Reporter Expression
      Additional Information
      Statement
      Reference
       
      Marker for
      Reflects expression of
      Reporter construct used in assay
      Human Disease Associations
      Disease Ontology (DO) Annotations
      Models Based on Experimental Evidence ( 0 )
      Disease
      Evidence
      References
      Modifiers Based on Experimental Evidence ( 0 )
      Disease
      Interaction
      References
      Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
       
      Disease-implicated variant(s)
       
      Phenotypic Data
      Phenotypic Class
      Phenotype Manifest In
      Detailed Description
      Statement
      Reference

      Clones in the eye reveal a phenotype qualitatively similar to that of ocuvi.

      (van den Dries et al., 1996)

      Neuromere b1 is absent or reduced in all embryos.

      (Hirth et al., 1995)

      Median ocellus is displaced anteriorly and is smaller. The pattern of interocellar and ocellar bristles is disturbed. The distance between the two lateral ocelli and between the two postvertical bristles is increased.

      (Royet and Finkelstein, 1995)

      No effect on prd expression in embryo.

      (Gutjahr et al., 1993)

      Clones in the head homozygous for ocotd-YH13 show abnormal morphology in the frons and ocelli. Clones in the female vaginal plates homozygous for ocotd-YH13 are always abnormal. Frequently the left and right plates are fused along the midline, and internal genital structures derived from the medial regions of the genital disc are missing. The thorn-like bristles on the vaginal plates are often duplicated. The first and fourth denticle rows in the anterior abdomen of ocotd-YH13 are deleted.

      (Wieschaus et al., 1992)

      Interacts with RpII140wimp maternal effect.

      (Parkhurst and Ish-Horowicz, 1991)

      The dorsal arms of the head skeleton are absent or fragmented in mutant embryos. The antennal segment is missing.

      (Finkelstein and Perrimon, 1990)

      Defects in head development and segmental patterning. Abnormal neuropil. Aberrant commissure formation in each segment. The disappearance of neurons labelled by P{A92}45C demonstrates that cell death is restricted to identified neurons associated with the midline of the CNS.

      (Finkelstein et al., 1990)

      All denticles of abdominal segments point posteriorly in ocotd-YH13 embryos. The head is abnormal.

      (Tearle and Nusslein-Volhard, 1987)
      External Data
      Interactions
      Show genetic interaction network for Enhancers & Suppressors
      Phenotypic Class
      Phenotype Manifest In
      Additional Comments
      Genetic Interactions
      Statement
      Reference
      Xenogenetic Interactions
      Statement
      Reference
      Complementation and Rescue Data
      Comments
      Images (0)
      Mutant
      Wild-type
      Stocks (2)
      Notes on Origin
      Discoverer
      Comments
      Comments

      ocotd-YH13 is partially dominant in combination with RpS5a2.

      (Wieschaus et al., 1992)

      Strong allele.

      (Gutjahr et al., 1993)
      External Crossreferences and Linkouts ( 0 )
      Synonyms and Secondary IDs (6)
      References (19)