Homozygous somatic clones in the follicle cells do not differentiate as polar cells, but block the differentiation of epithelial follicle cells and causes them to remain as undifferentiated precursors. When a clone falls in a region where the follicle cells normally undergo morphogenetic movement, these movements do not occur. The stretched follicle cells fail to move over the nurse cells; the centripetal cells do not migrate between the oocyte and the nurse cells; and the more posterior follicle cells fail to move over the oocyte. No somatic clones in the stalk cells or the polar follicle cells are recovered. Occasionally, clones in the follicle cells leads to a failure of cyst encapsulation leading to fused or partially fused egg chambers. Homozygous mutant clones in the epithelial follicle cells are similar in size to wild-type twin spots, but contain many more smaller cells: the cells go through extra cell divisions, without a corresponding increase in growth rate. In wild type cells division ceases at stage 6, however mutant cells continue to divide up stages 10B or 11. Furthermore these cells have a lower DNA content indicating that mutant cells fail to switch from the mitotic cell cycle to the endo cycle and carry on dividing instead of becoming polyploid.

Homozygous clones in the antenna result in the complete absence of bristles and a smaller antenna than normal. Cells that comprise the sensilla fail to develop in antenna clones, in contrast to clones recovered elsewhere on the body that result in the absence of bristles accompanied by a proliferation of sensory neurons at the expense of cuticular structures.