Expression of lin^Scer\UAS.cHa under the control of Scer\GAL4^{Ubx-lac1-Gal4} results in flies lacking both heminota.

Expression of lin^Scer\UAS.cHa under the control of Scer\GAL4^Act5C.PU in large clones induced during the first larval instar results in flies lacking a heminotum.

Expression of lin^Scer\UAS.cHa under the control of Scer\GAL4^klu-G410 results in a reduction in the extent of the prescutum and eliminates lateral microchaetae in the ventral prescutum.

Larvae expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^dpp.blk1 show antennal duplication in both antennal discs and pharate adults, or formation of ventral eyelets in the head capsule.

The wing discs have a spherical morphology in animals expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^{Ubx-lac1-Gal4}. The thin peripodial epithelium is replaced with a mirror image duplication of the thickened, pseudostratified columnar epithelium that forms the disc proper. The mutant discs are approximately 10-30% the size of wild-type wing discs. The number of actively dividing cells in the mutant discs is similar to wild type, but the number of apoptotic cells is elevated in the mutant discs. The mutant discs survive beyond the larval stages and elongated during metamorphosis, but fail to contribute to adult structures.

Large clones in the disc proper of the wing disc that are expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^Act5C have no effect on wing development.

Large clones in the peripodial epithelium of the wing disc that are expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^Act5C completely transform the peripodial epithelium into disc proper tissue, resulting in a mirror image duplication of the pseudostratified columnar epithelium. These discs are spherical and smaller than normal.

Smaller clones in the peripodial epithelium of the wing disc that are expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^Act5C result in the formation of secondary wing fields in the discs. The presumptive notum appears intact in some of these discs, but is reduced or missing in others. The clones that produce this phenotype adopt disc proper fate and occupy approximately 26% of the surface area of the entire wing disc.

Peripodial epithelium cells in the wing disc that are expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^ptc-559.1 adopt disc proper fate and permit the induction of a secondary wing field in the disc.

Clones at the margin in the eye disc that are expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^Act5C.PP result in a failure of retinal initiation.

The 1^o-3^o fates of the dorsal epidermis are replaced with the 4^o fate in embryos expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^ptc-559.1.

Embryos expressing lin^Scer\UAS.cHa under the control of Scer\GAL4^byn-Gal4 have a short, wide hindgut, consisting of an undulating layer of columnar cells with a duplication of the boundary cell rows.

Expression of lin^Scer\UAS.cHa under the control of Scer\GAL4^arm.PS results in replacement of 1^o-3^o fates in the dorsal cuticle (dorsal denticles, smooth cuticle and thick hairs respectively) with 4^o fates (fine hairs). Expression of lin^Scer\UAS.cHa under the control of Scer\GAL4^ptc-559.1 is occasionally not sufficient to specify the 4^o dorsal cuticle fate (fine hairs) in place of the 2^o fate (smooth cuticle). Expression of lin^Scer\UAS.cHa under the control of Scer\GAL4^en-e16E in lin^G2/lin² embryos transforms all en-expressing cells to the 4^o fate (fine hairs).