Scer\FRT-mediated recombination between the two progenitor insertions has resulted in the deletion of the genomic sequence between them.
ISNb axons of homozygous Df(2R)A15 mutant embryos show frequent defasciculation defects at muscle 12 and innervation defects at muscles 6/7, but not ectopic innervations, as compared to controls. SNa axons of homozygous Df(2R)A15 mutant embryos sometimes fail to innervate muscle 24, resulting in a 'stall' phenotype, as compared to controls.
This deficiency includes a haploinsufficient locus, manifest as a dominant male sterility phenotype.
This deficiency includes a haploinsufficient region, and thus is maintained in a stable stock by the inclusion of PBac{IbaS}, which covers this region.
76.0% of hemisegments show disorganisation of the intermediate Fas2-positive longitudinal tract in homozygous embryos carrying PBac{IbaS}. The terminal branches of chordotonal organ axons in the central nervous system are completely disorganised, showing both ectopic lateral and medial projections.