Spermatogenesis in S-Lap1Δ7 homozygotes shows normal cell divisions, normal nebenkern number and formation, normal length of elongated cysts, normal establishment of individualization complexes around the needle shaped nuclei, and normal number of total individualization complexes; however, individualization migrate less frequently and more asynchronously than controls, the associated major mitochondrial derivatives show structural defects (investment cones are dispersed but DJ-GFP distribution is slightly dashed; ). As in controls, elongated spermatids shows normal axoneme structures, there is complete formation of the two mitochondrial derivatives, and paracrystalline material start accumulating in the major mitochondrial derivative, although the major mitochondrial derivative has abnormal size and the paracrystalline material is disorganized/dispersed.
S-Lap3MB01319 has male sterile | recessive phenotype, non-suppressible by S-Lap1+tLa
S-Lap3MB01319 has male sterile | recessive phenotype, non-suppressible by S-Lap1int.Tag:HA
S-Lap3[+]/S-Lap3MB01319 is an enhancer of male semi-sterile | dominant phenotype of S-Lap1Δ7
S-Lap1Δ7, S-Lap3[+]/S-Lap3MB01319 has male semi-sterile | dominant phenotype
S-Lap3MB01319, S-Lap5[+]/S-Lap5Δ14 has male semi-sterile | dominant phenotype
S-Lap3MB01319, loopin-1[+]/loopin-1MI06848 has male semi-sterile | dominant phenotype
S-Lap3MB01319, S-Lap4[+]/S-Lap4MB11296 has male semi-sterile | dominant phenotype
S-Lap2MI14597/+, S-Lap3MB01319/+ males yield fewer offspring than controls.