Deletion that removes part of Nost; almost all the protein-coding exons of each isoform are deleted. Generated by FLP-mediated recombination between the PBac{WH}f06363 and P{XP}Nostd08142 insertions.
Recombination between the progenitor insertions results in a deletion that removes almost all Nost exons.
Recombination between the progenitor insertions results in a deletion that removes most of the Nost CDS.
Deletion that removes part of Nost; almost all the protein-coding exons of each isoform are deleted. Generated by Scer\FLP1-mediated recombination between the PBac{WH}f06363 and P{XP}Nostd08142 insertions.
NostΔ mutants have no obvious phenotypes.
Cip4Δ32, Nostdf004 has partially lethal - majority die phenotype
Cip4Δ32, Nostdf004 has female sterile phenotype
Cip4Δ32, Nostdf004 has male semi-sterile phenotype
Nostdf004/Nostdf004 is an enhancer of adult midgut longitudinal muscle cell phenotype of Cip4Δ32
Nostdf004/Nostdf004 is an enhancer of adult midgut circular muscle cell phenotype of Cip4Δ32
Nostdf004 is an enhancer of wing hair | increased number phenotype of Cip4Δ32
Nostdf004 is an enhancer of egg chamber phenotype of Cip4Δ32
Cip4Δ32, Nostdf004, SyndΔEx22 has embryonic/larval midgut | embryonic stage phenotype
Cip4Δ32, Nostdf004, SyndΔEx22 has embryonic somatic muscle cell phenotype
Cip4Δ32, Nostdf004, SyndΔEx22 has muscle cell of dorsal acute muscle 1 | embryonic stage phenotype
Cip4Δ32, Nostdf004 has embryonic somatic muscle cell phenotype
Cip4Δ32, Nostdf004 has muscle cell of dorsal acute muscle 1 | embryonic stage phenotype
Cip4Δ32 Nostdf004 maternal and zygotic homozygous double mutant embryos exhibit somatic muscle defects (missing or small muscle fibers in several segments replaced with mononucleated myoblasts at the corresponding positions) and significantly decreased number of nuclei in muscle DA1 when compared to controls.
Nostdf004 maternal and zygotic Cip4Δ32 zygotic SyndΔEx22 zygotic triple homozygous mutant embryos exhibit somatic muscle defects (missing muscle fibers with unfused myoblasts), significantly decreased number of nuclei in muscle DA1 and slightly expanded midgut anterior chamber when compared to controls.
44.8% of Cip4Δ32 NostΔ ovaries have defective egg chambers containing an aberrant number of germline cells - they always have two or more oocytes, each oocyte has four ring canals, and oocytes are randomly positioned, indicating a defect in the encapsulation of single germline cysts by follicle cells. Defective egg chambers past stage 9 are not observed. Germarium morphology is clearly affected.