The climbing initiation behaviour and gap-crossing success of sev1 flies is not different from wild-type flies.
R8 cells occupy the vacant R7 position. Several facets are missing R8 and R7 cells.
Ommatidia of homozygotes have no central rhabdomeres. Ommatidia of double mutants with aoppok-1 contain a variable number of R7-like cells.
Absence of R7, insensitivity to UV-light.
Inner rhabdomere seven is missing.
Flies lack R7 receptor cells in every ommatidium. Flies show positive phototaxis (similar to wild-type) in response to extremely dim light stimuli.
negative phototaxis with Ppb/+
sev1 has abnormal visual behavior phenotype, suppressible by Ppb[+]/Ppb1
sev1 has abnormal phototaxis phenotype, suppressible by Ppb[+]/Ppb1
Rh52, Rh61, sev1 has abnormal optomotor response phenotype
sev1, sl1 has photoreceptor cell R7 phenotype
sev1, sl2 has photoreceptor cell R7 phenotype
SosJC2 does not suppress the mutant phenotype, R7 cells are absent from ommatidia.
sev1 mutants - i.e. sev1; Ppb1/+ flies, like wild-type flies, prefer UV over green light, while sev1;+/+ flies prefer green over UV. When tested in a T-maze for preference between darkness and green light (550 nm), both wild type and sev1 flies show positive phototaxis, while sev1;Ppb1/+ and sev1;Ppb1/Ppb1 flies show negative phototaxis. Similarly, tests in a countercurrent apparatus for movement toward and movement away from white light indicate marked preference of wild-type flies for light and of sev1;Ppb1/+ flies for darkness. Both the positive phototactic response of sev1 and the negative phototactic response of sev1;Ppb1/+ flies increase with age, reaching a maximum at 4-6 days. The interaction between Ppb and sev is allele-specific, as is indicated in the sev allele entries. Photoreceptor cell R7, missing in all of the sev alleles, was not restored in any mutant combination of Ppb and sev.
Harris.
Normal orientation effects in the fixation Y-maze.