Expression of UbxScer\UAS.cMa under the control of Scer\GAL4Vap.P0201 results in only 8% rescue of the cell death that is normally seen in anterior dMP2 neurons in late embryos. Expression of UbxScer\UAS.cMa under the control of Scer\GAL4elav-C155 only results in a marginal increase in survival of anterior dMP2 neurons in late embryos compared to wild-type embryos (where these neurons are lost by the late embryonic stage).
Expression of UbxScer\UAS.cMa under the control of Scer\GAL4sca-537.4 results in a mutant phenotype in the embryonic tritocerebrum. The phenotype has a penetrance of more than 95%.
Expression of UbxScer\UAS.cMa under the control of Scer\GAL4lab.PH does not result in morphological defects in the tritocerebrum or any other part of the embryonic brain.
When driven by Scer\GAL4how-24B, shows a clear transformation of posterior pharyngeal wall to an abdominal character. Two distinct rows of abdominal denticles are found on this structure. Prominent posterior central projections (PVs) are seen in the entire thorax indicating a partial transformation towards abdominal identity.
When driven by Scer\GAL469B, Scer\GAL4prd.RG1, or Scer\GAL4l(3)31-1-31-1 leads to a significant loss in viability.
Scer\GAL4lab.PH/UbxUAS.cMa is a suppressor of embryonic tritocerebrum phenotype of lab14
Expression of UbxScer\UAS.cMa under the control of Scer\GAL4lab.PH rescues the tritocerebral defects seen in lab14 embryonic brains. 32.6% of embryos show a complete rescue of the defects (taking into account that the phenotypic penetrance of the lab14 phenotype is 88.6%).