Mutant adults show only a transient electroretinogram response to light, in contrast to the sustained response seen in wild-type flies.
trpMB03672 has abnormal neurophysiology | adult stage phenotype, non-enhanceable by Xport-A1
trpMB03672 has abnormal neurophysiology | adult stage phenotype, non-enhanceable by Xport-BΔB
trpMB03672 has abnormal neurophysiology | adult stage phenotype, non-enhanceable by Df(3R)Xport-ΔAB
trpMB03672 has abnormal neurophysiology | adult stage phenotype, non-suppressible by Xport-A1
trpMB03672 has abnormal neurophysiology | adult stage phenotype, non-suppressible by Xport-BΔB
trpMB03672 has abnormal neurophysiology | adult stage phenotype, non-suppressible by Df(3R)Xport-ΔAB
trpMB03672/trpMB03672 is a suppressor of abnormal circadian rhythm | adult stage phenotype of cry01, norpAP24
Additional homozygosity for trpMB03672 rescues the circadian rhythmicity defects observed in cry01, norpA36 double homozygous adults previously trained under 12h:12h light:dark cycles.
The electroretinogram defects seen in trpMB03672 flies are not altered if the flies are also mutant for one of Xport-A1, Xport-BΔB or Df(3R)Xport-ΔAB.