UASt regulatory sequences drive expression of a 500bp inverted repeat.
Larvae expressing Hr46NIG.12208R under the control of Scer\GAL4phm.PO in the prothoracic glands fail to pupariate on time, continuing to feed and grow.
Hr3NIG.12208R, Scer\GAL4phtm.PO has abnormal developmental rate | larval stage phenotype, non-enhanceable by Hr3NIG.12208R, Scer\GAL4phtm.PO
Hr3NIG.12208R, Scer\GAL4phtm.PO has abnormal developmental rate | larval stage phenotype, non-suppressible by Hr3NIG.12208R, Scer\GAL4phtm.PO
Hr3NIG.12208R, Scer\GAL4phtm.PO is a non-enhancer of abnormal developmental rate | larval stage phenotype of Hr3NIG.12208R, Scer\GAL4phtm.PO
Hr3NIG.12208R, Scer\GAL4phtm.PO is a non-enhancer of abnormal developmental rate | larval stage phenotype of NosRNAi.UAS.cCa, Scer\GAL4phtm.PO
Hr3NIG.12208R, Scer\GAL4phtm.PO is a non-suppressor of abnormal developmental rate | larval stage phenotype of Hr3NIG.12208R, Scer\GAL4phtm.PO
Hr3NIG.12208R, Scer\GAL4phtm.PO is a non-suppressor of abnormal developmental rate | larval stage phenotype of NosRNAi.UAS.cCa, Scer\GAL4phtm.PO
Coexpression of Hr46NIG.12208R with NosdsRNA.Scer\UAS.IR-X in prothoracic glands (with both transgenes under the control of Scer\GAL4phm.PO) results in prolonged larval growth, as in the single mutants.