UAS regulatory sequences drive expression of an inverted repeat.
Expression of EcRJF02538 in the fat body under the control of Scer\GAL4DcG results in significant hyperglycaemia.
Scer\GAL4esg.PU, EcRJF02538 is a suppressor of abnormal cell cycle | male | adult stage phenotype of DpUAS.cDa, E2f1UAS.cNa, Scer\GAL4esg.PU
EcRJF02538, Scer\GAL4esg.PU is a non-suppressor of abnormal cell cycle | adult stage | virgin male phenotype of CycEUAS.cRa, Scer\GAL4esg.PU
EcRJF02538, Scer\GAL4esg.PU is a non-suppressor of increased cell growth rate | adult stage | virgin male phenotype of CycEUAS.cRa, Scer\GAL4esg.PU
Scer\GAL4esg.PU, EcRJF02538 is a suppressor of male accessory gland secondary cell phenotype of DpUAS.cDa, E2f1UAS.cNa, Scer\GAL4esg.PU
EcRJF02538, Scer\GAL4esg.PU is a non-suppressor of male accessory gland secondary cell | virgin male phenotype of CycEUAS.cRa, Scer\GAL4esg.PU
Expression of EcRJF02538 under the control of Scer\GAL4mir-310.PL in Df(2R)Dm310s mutant background almost fully rescues the egg dorsal appendages defect characteristic for the Df(2R)Dm310s mutant eggs but it further exacerbates their reduced hatchability as well as the decreased male fertility (assayed under sperm-depletion conditions).